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Office Location: 226 SB
Mail Code: 179
Phone: (801) 863-7031
Fax: (801) 863-8064
Department: Biology
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Academic Record:
Brigham Young University, Provo, UT, Ph.D. Emphasis: Cell Biology and Physiology, 1999
Brigham Young University, Provo, UT, B.A. Magna Cum Laude,1994
Professional Experience:
Assistant Professor, Department of Biology, Utah Valley State College, Orem, UT , Sept. 2000-present
Visiting Professor, Department of Zoology, Brigham Young University, Provo, UT , Jan 2000-Aug 2000
Professional Societies:
Biophysical Society Member, 1997-present
American Society for Cell Biology, 1999-2003
Honors:
UVSC Foundation Grant, Utah Valley State College, May 2003, 2002
Nominated for Teacher of the Year, Utah Valley State College, Spring 2002
Presidential award for faculty scholarship, Utah Valley State College, Sept. 2002
Pope Institute Research Grant, Utah Valley State College, May 2001
Publications:
Wilson, H.A., Allred, D.V., O?Neill, K., and J.D. Bell. (2000) Activities and interactions among phospholipases A2 during thapsigargin-induced S49 cell death. Apoptosis, 5, 389-396.
Wilson, H.A., Waldrip, J.B., Nielson, K.H., Judd, A.M., Koduri, R.S., Gelb, M.H., Han, A.K., Cho. W., Sims, P.J., and J.D. Bell. (1999) Mechanisms by which elevated intracellular calcium induces S49 cell membranes to become susceptible to the action of secretory phospholipase A2. Journal of Biological Chemistry , 274, 11494-11504.
Wilson, H.A., Huang, W., Waldrip, J.B., Judd, A.M., Vernon, L.P., and J.D. Bell. (1997) Mechanisms by which thionin induces membrane susceptibility of S49 cell membranes to extracellular phospholipase A2. Biochimica et Biophysica Acta, 1349, 142-156.
Winder, W.W., Wilson, H.A., Hardie, D.G., Rasmussen, B.B., Hutber, C.A., Call. G.B., Clayton, R.D., Conley, L.M., Yoon, S., and Zhou, B. (1997) Phosphorylation of rat muscle acetyl-CoA carboxylase by AMP-activated protein kinase and protein kinase A. Journal of Applied Physiology, 82(1), 219-225.
Abstracts:
Wilson, H.A., Judd, A.M., and Bell, J.D. (2003) Mechanism by which phospholipase A2 causes cells to become resistant to its own action. FASEB J. 17:A578.
Wilson, H.A., and Bell, J.D. (2002) Formation of transient non-protein calcium channels by lysophospholipid in S49 lymphoma cells. FASEB J. 16:A906.
Wilson, H.A. and J.D.Bell. (2002) Secretory phospholipase A2 causes mammalian cell membranes to become refractory to further hydrolysis by the enzyme. Biolphys. J. 80, 549a.
Wilson, H.A. and J.D.Bell. (2001) Secretory phospholipase A2 causes mammalian cell membranes to become refractory to further hydrolysis by the enzyme. Biolphys. J. 80, 549a.
Wilson, H.A., Allred, D.V., O?Neill, K.L., and J.D. Bell. (1999) Roles of intracellular phospholipase A2 in the induction of membrane susceptibility to sPLA2 during apoptosis. Mol. Bio. Cell 10, 189a (#1093).
Wilson, H.A., and J.D. Bell. (1998) Novel cellular response to lysophosphatidylcholine: calcium-dependent changes in membrane structure and susceptibility to phospholipase A2. Mol. Bio. Cell 9, 80a (#464).
Wilson, H.A., Olsen, C.A., and J.D. Bell. (1997) Induction of susceptibility to secretory phospholipase A2 in S49 lymphoma cells. Mol. Bio. Cell 8, 78a (#456).
Wilson, H.A., Judd, A.M., and J.D. Bell. (1997) Mechanisms by which thionin induces susceptibility of S49 cell membranes to phospholipase A2. Biophys. J. 70, A419 (#W-Pos234).
Invited Presentations
Wilson, H.A., Judd, A.M., and J.D. Bell. (2003) Mechanism by which phospholipase A2 causes cells to become resistant to its action. FASEB Conference, San Diego CA.
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